JImmunol[Jour]AND[volume]AND1[issue]
免疫学杂志卷1期
1
CCL17inInflammationandPain.
CCL17用于炎症和疼痛。
LeeKM,JarnickiA,AchuthanA,FleetwoodAJ,AndersonGP,EllsonC,FeeneyM,ModisLK,SmithJE,HamiltonJA,CookA.
JImmunol.Jul1;(1):-.
2
TracingSelf-ReactiveBCellsinNormalMice.
追踪正常小鼠自身反应性B细胞。
NojimaT,ReynoldsAE,KitamuraD,KelsoeG,KuraokaM.
JImmunol.Jul1;(1):90-.
3
MerocyticDendriticCellsComposeaConventionalDendriticCellSubsetwithLowMetabolicActivity.
红细胞树突状细胞是一种代谢活性低的传统树突状细胞亚群。
AudigerC,FoisA,ThomasAL,JanssenE,PelletierM,LesageS.
JImmunol.Jul1;(1):-.
4
PorphyromonasgingivalisPromotesImmunoevasionofOralCancerbyProtectingCancerfromMacrophageAttack.
牙龈卟啉单胞菌通过保护癌细胞免受巨噬细胞攻击促进口腔癌免疫逃避。
LiuS,ZhouX,PengX,LiM,RenB,ChengG,ChengL.
JImmunol.Jul1;(1):-.
5
TopReads.
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JImmunol.Jul1;(1):1.
6
RequirementofComplementC6forIntactInnateImmuneResponsesinMice.
补体C6对小鼠完整天然免疫反应的需求。
FattahiF,GrailerJJ,ParlettM,LuH,MalanEA,AbeE,RussellMW,FrydrychLM,DelanoMJ,ZetouneFS,WardPA.
JImmunol.Jul1;(1):-.
7
CharacterizationofDNABindingandNuclearRetentionIdentifiesZebrafishIRF11asaPositiveRegulatorofIFNAntiviralResponse.
DNA结合和核保留的特征确定斑马鱼IRF11是干扰素抗病毒反应的阳性调节因子。
ZhaoX,GongXY,LiYL,DanC,GuiJF,ZhangYB.
JImmunol.Jul1;(1):-.
8
UpregulatedPKM2inMacrophagesExacerbatesExperimentalArthritisviaSTAT1Signaling.
巨噬细胞中PKM2上调通过STAT1信号加重实验性关节炎。
XuJ,JiangC,WangX,GengM,PengY,GuoY,WangS,LiX,TaoP,ZhangF,HanY,NingQ,ZhuW,MengL,LuS.
JImmunol.Jul1;(1):-.
9
ImmuneComplex-DrivenGenerationofHumanMacrophageswithAnti-InflammatoryandGrowth-PromotingActivity.
免疫复合物驱动产生具有抗炎和促生长活性的人巨噬细胞。
DalbyE,ChristensenSM,WangJ,HamidzadehK,ChandrasekaranP,HughittVK,TafuriWL,ArantesRME,RodriguesIA,HerbstR,El-SayedNM,SimsGP,MosserDM.
JImmunol.Jul1;(1):-.
10
COVID-19asanAcuteInflammatoryDisease.
COVID-19是一种急性炎症性疾病。
ManjiliRH,ZareiM,HabibiM,ManjiliMH.
JImmunol.Jul1;(1):12-19.
11
CordBloodLow-DensityGranulocytesCorrespondtoanImmatureGranulocyticSubsetwithLowExpressionofSA12.
脐血低密度粒细胞对应于低表达SA12的未成熟粒细胞亚群。
WeinhageT,K?lscheT,Rieger-FackeldeyE,SchmitzR,AntoniAC,AhlmannM,FoellD,WittkowskiH.
JImmunol.Jul1;(1):56-66.
12
TRIM14IsaKeyRegulatoroftheTypeIIFNResponseduringMycobacteriumtuberculosisInfection.
TRIM14是结核分枝杆菌感染时I型干扰素应答的关键调节因子。
HoffpauirCT,BellSL,WestKO,JingT,WagnerAR,Torres-OdioS,CoxJS,WestAP,LiP,PatrickKL,WatsonRO.
JImmunol.Jul1;(1):-.
13
AssociationbetweenSubcutaneousAdiposeTissueInflammation,InsulinResistance,andCalorieRestrictioninObeseFemales.
肥胖女性皮下脂肪组织炎症、胰岛素抵抗和热量限制的关系。
Sbierski-KindJ,MaiK,KathJ,JurischA,StreitzM,KuchenbeckerL,BabelN,NienenM,JürchottK,SprangerL,JumpertzvonSchwartzenbergR,DeckerAM,KrügerU,VolkHD,SprangerJ.
JImmunol.Jul1;(1):45-55.
14
NewInsightsintotheImmuneSystemUsingDirtyMice.
用脏老鼠研究免疫系统的新发现。
HamiltonSE,BadovinacVP,BeuraLK,PiersonM,JamesonSC,MasopustD,GriffithTS.
JImmunol.Jul1;(1):3-11.
15
LossofPhosphatidylinositol3-KinaseActivityinRegulatoryTCellsLeadstoNeuronalInflammation.
调节性T细胞中磷脂酰肌醇3激酶活性的丧失导致神经元炎症。
StarkAK,DavenportECM,PattonDT,ScudamoreCL,VanhaesebroeckB,VeldhoenM,GardenOA,OkkenhaugK.
Version2.JImmunol.Jul1;(1):78-89.
16
HLAClassIISpecificityAssessedbyHigh-DensityPeptideMicroarrayInteractions.
高密度肽微阵列相互作用评估HLAⅡ类特异性。
OsterbyeT,NielsenM,DudekNL,RamarathinamSH,PurcellAW,Schafer-NielsenC,BuusS.
JImmunol.Jul1;(1):-.
17
TriggeringofthecGAS-STINGPathwayinHumanPlasmacytoidDendriticCellsInhibitsTLR9-MediatedIFNProduction.
触发人浆细胞样树突状细胞cGAS-STING通路抑制TLR9介导的IFN产生。
DebP,DaiJ,SinghS,KalyoussefE,Fitzgerald-BocarslyP.
JImmunol.Jul1;(1):-.
18
PlacentalMyeloidCellsProtectagainstZikaVirusVerticalTransmissioninaRag1-DeficientMouseModel.
在Rag1缺陷小鼠模型中胎盘髓细胞对寨卡病毒垂直传播的保护作用。
WinklerCW,EvansAB,CarmodyAB,PetersonKE.
JImmunol.Jul1;(1):-.
19
Opsonization-EnhancedAntigenPresentationbyMR1ActivatesRapidPolyfunctionalMAITCellResponsesActingasanEffectorArmofHumoralAntibacterialImmunity.
MR1的调理增强抗原提呈激活多功能MAIT细胞快速反应,作为体液抗菌免疫的效应臂。
BoulouisC,GorinJB,DiasJ,BergmanP,LeeansyahE,SandbergJK.
Version2.JImmunol.Jul1;(1):67-77.
20
ExpansionofGroup2InnateLymphoidCellsinPatientswithEnd-StageRenalDiseaseandTheirClinicalSignificance.
终末期肾病患者2组固有淋巴细胞扩增及其临床意义。
LiuGY,DengXH,LiX,CaoYJ,XingYF,ZhouP,LeiAH,YangQ,DengK,ZhangH,ZhouJ.
JImmunol.Jul1;(1):36-44.
21
EarlyTCellActivationMetricsPredictGraft-versus-HostDiseaseinaHumanizedMouseModelofHematopoieticStemCellTransplantation.
早期T细胞活化指标预测人源化造血干细胞移植小鼠移植物抗宿主病。
HessNJ,HudsonAW,HemattiP,GumperzJE.
JImmunol.Jul1;(1):-.
22
SUMOylationofDDX39AAltersBindingandExportofAntiviralTranscriptstoControlInnateImmunity.
DDX39A的SUMOylation改变抗病毒转录物的结合和输出以控制先天免疫。
ShiP,GuoY,SuY,ZhuM,FuY,ChiH,WuJ,HuangJ.
JImmunol.Jul1;(1):-.
23
IgANephropathyBenefitsfromCompoundKTreatmentbyInhibitingNF-κB/NLRP3InflammasomeandEnhancingAutophagyandSIRT1.
IgA肾病通过抑制NF-κB/NLRP3炎症小体,增强自噬和SIRT1而受益。
WuCY,HuaKF,HsuWH,SuzukiY,ChuLJ,LeeYC,TakahataA,LeeSL,WuCC,Nikolic-PatersonDJ,KaSM,ChenA.
JImmunol.Jul1;(1):-.
24
MHC-IndependentThymicSelectionofCD4andCD8CoreceptorNegativeαβTCells.
CD4和CD8共受体阴性αβT细胞的MHC非依赖性胸腺选择。
CollinR,Lombard-VadnaisF,HillhouseEE,LebelMè,Chabot-RoyG,MelicharHJ,LesageS.
JImmunol.Jul1;(1):-.
25
MaintenanceofGerminalCenterBCellsbyCaspase-9throughPromotionofApoptosisandInhibitionofNecroptosis.
Caspase-9通过促进凋亡和抑制坏死下垂维持生发中心B细胞。
ZhangJ,KodaliS,ChenM,WangJ.
JImmunol.Jul1;(1):-.
26
CuttingEdge:STAT1-MediatedEpigeneticControlofRsad2PromotesClonalExpansionofAntiviralNKCells.
前沿:STAT1介导的Rsad2表观遗传控制促进抗病毒NK细胞的克隆扩增。
WiedemannGM,GearyCD,LauCM,SunJC.
JImmunol.Jul1;(1):21-25.
27
CriticalRoleofTSLPReceptoronCD4TCellsforExacerbationofSkinInflammation.
CD4T细胞表面TSLP受体在皮肤炎症加重中的作用。
KitajimaM,KuboM,ZieglerSF,SuzukiH.
JImmunol.Jul1;(1):27-35.
28
ThermalBurnInjuryGeneratesBioactiveMicrovesicles:EvidenceforaNovelTransportMechanismfortheLipidMediatorPlatelet-ActivatingFactor(PAF)ThatInvolvesSubcellularParticlesandthePAFReceptor.
热烧伤产生生物活性微泡:脂质介质血小板活化因子(PAF)新的转运机制的证据,涉及亚细胞颗粒和PAF受体。
LiuL,FahyKE,AwoyemiAA,ThapaP,KellyLE,ChenJ,BihlJC,CoolDR,ChenY,RappCM,JohnsonRM,TraversJB.
JImmunol.Jul1;(1):-.
29
DirectAntiviralActivityofIFN-StimulatedGenesIsResponsibleforResistancetoParamyxovirusesinISG15-DeficientCells.
IFN刺激基因的直接抗病毒活性与ISG15缺陷细胞对副粘病毒的抵抗有关。
HolthausD,VasouA,BamfordCGG,AndrejevaJ,PaulusC,RandallRE,McLauchlanJ,HughesDJ.
Version2.JImmunol.Jul1;(1):-.
30
Correction:HepatitisBVirusSurfaceAntigenEnhancestheSensitivityofHepatocytestoFas-MediatedApoptosisviaSuppressionofAKTPhosphorylation.
纠正:乙型肝炎病毒表面抗原通过抑制AKT磷酸化增强肝细胞对Fas介导的凋亡的敏感性。
JingZT,LiuW,WuSX,HeY,LinYT,ChenWN,LinXJ,LinX.
JImmunol.Jul1;(1):-.
JImmunol[Jour]AND[volume]AND2[issue]
免疫学杂志卷2期
1
LossofIL-27RαResultsinEnhancedTubulointerstitialFibrosisAssociatedwithElevatedTh17Responses.
白细胞介素-27Rα的丢失导致肾小管间质纤维化增强,与Th17反应升高相关。
CoppockGM,AronsonLR,ParkJ,QiuC,ParkJ,DeLongJH,RadaelliE,SusztákK,HunterCA.
JImmunol.Jul15;(2):-.
2
TopReads.
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JImmunol.Jul15;(2):-.
3
HypercapniaSuppressesMacrophageAntiviralActivityandIncreasesMortalityofInfluenzaAInfectionviaAkt1.
高碳酸血症抑制巨噬细胞抗病毒活性,并通过Akt1增加甲型流感感染的死亡率。
Casalino-MatsudaSM,ChenF,Gonzalez-GonzalezFJ,NairA,DibS,YemelyanovA,GatesKL,BudingerGRS,BeitelGJ,SpornPHS.
JImmunol.Jul15;(2):-.
4
LeukocyteTetraspaninCD53Restrainsα3IntegrinMobilizationandFacilitatesCytoskeletalRemodelingandTransmigrationinMice.
白细胞四肽CD53抑制α3整合素动员,促进小鼠细胞骨架重构和迁移。
YeungL,AndersonJML,WeeJL,DemariaMC,FinsterbuschM,LiuYS,HallP,SmithBC,DankersW,ElgassKD,WicksIP,KwokHF,WrightMD,HickeyMJ.
JImmunol.Jul15;(2):-.
5
IL-6DeficiencyExacerbatesAllergicAsthmaandAbrogatestheProtectiveEffectofAllergicInflammationagainstStreptococcuspneumoniaePathogenesis.
IL-6缺乏可加重过敏性哮喘,消除过敏性炎症对肺炎链球菌发病的保护作用。
SchmitT,GhoshS,MathurRK,BarnhardtT,AmbigapathyG,WuM,CombsC,KhanMN.
JImmunol.Jul15;(2):-.
6
CellType-andStimulation-DependentTranscriptionalProgramsRegulatedbyAtg16L1andItsCrohnsDiseaseRiskVariantTA.
Atg16L1及其克罗恩病风险变异体TA调控的细胞类型和刺激依赖性转录程序。
VarmaM,KadokiM,LefkovithA,ConwayKL,GaoK,MohananV,TusiBK,GrahamDB,LatorreIJ,TolonenAC,KhorB,NgA,XavierRJ.
JImmunol.Jul15;(2):-.
7
InfluenzaSuppressesNeutrophilRecruitmenttotheLungandExacerbatesSecondaryInvasivePulmonaryAspergillosis.
流感抑制中性粒细胞向肺的募集,加重继发性侵袭性肺曲菌病。
TobinJM,NickolichKL,RamananK,PilewskiMJ,LamensKD,AlcornJF,RobinsonKM.
JImmunol.Jul15;(2):-.
8
UpregulationofProinflammatoryBradykininPeptidesinSystemicLupusErythematosusandRheumatoidArthritis.
系统性红斑狼疮和类风湿关节炎中促炎性缓激肽的上调。
VanarsaK,HendersonJ,SoomroS,QinL,ZhangT,JordanN,PuttermanC,BlancoI,SaxenaR,MohanC.
JImmunol.Jul15;(2):-.
9
CuttingEdge:TNFIsEssentialforMycobacteria-InducedMINCLEExpression,MacrophageActivation,andTh17Adjuvanticity.
尖端:TNF对分枝杆菌诱导的MINCLE表达、巨噬细胞活化和Th17佐剂作用至关重要。
SchickJ,Sch?ferJ,AlexanderC,DichtlS,MurrayPJ,ChristensenD,SorgU,PfefferK,SchleicherU,LangR.
JImmunol.Jul15;(2):-.
10
RoleofAgingandtheImmuneResponsetoRespiratoryViralInfections:PotentialImplicationsforCOVID-19.
衰老和呼吸道病毒感染的免疫反应:对COVID-19的潜在影响。
ChenJ,KelleyWJ,GoldsteinDR.
JImmunol.Jul15;(2):-.
11
UpregulationofMultipleCD8+TCellExhaustionPathwaysIsAssociatedwithRecurrentOcularHerpesSimplexVirusType1Infection.
多条CD8+T细胞衰竭途径上调与复发性眼部单纯疱疹病毒1型感染相关。
CoulonPG,RoyS,PrakashS,SrivastavaR,DhanushkodiN,SalazarS,AmezquitaC,NguyenL,VahedH,NguyenAM,WarsiWR,YeC,Carlos-CruzEA,MaiUT,BenMohamedL.
JImmunol.Jul15;(2):-.
12
ImpactofCysteineResiduesonMHCBindingPredictionsandRecognitionbyTumor-ReactiveTCells.
半胱氨酸残基对肿瘤反应性T细胞MHC结合预测和识别的影响。
SachsA,MooreE,Kosaloglu-YalcinZ,PetersB,SidneyJ,RosenbergSA,RobbinsPF,SetteA.
JImmunol.Jul15;(2):-.
13
AccessibilityofOAntigensSharedbetweenSalmonellaSerovarsDeterminesAntibody-MediatedCross-Protection.
血清型沙门氏菌之间共享的O抗原的可及性决定了抗体介导的交叉保护。
AhmedA,AkhadeAS,QadriA.
JImmunol.Jul15;(2):-.
14
VitaminDRegulatesMerTK-DependentPhagocytosisinHumanMyeloidCells.
维生素D调节人髓系细胞的MerTK依赖性吞噬功能。
ClarkeJ,YaqubiM,FutheyNC,SedaghatS,BaufeldC,BlainM,BaranziniS,ButovskyO,AntelJ,WhiteJH,HealyLM.
JImmunol.Jul15;(2):-.
15
InflammasomesandPyroptosisasTherapeuticTargetsforCOVID-19.
炎症小体和上睑下垂作为COVID-19的治疗靶点。
YapJKY,MoriyamaM,IwasakiA.
JImmunol.Jul15;(2):-.
16
P2Y6DeficiencyEnhancesDendriticCell-MediatedTh1/Th17DifferentiationandAggravatesExperimentalAutoimmuneEncephalomyelitis.
P2Y6缺乏增强树突状细胞介导的Th1/Th17分化,加重实验性自身免疫性脑脊髓炎。
LiZ,HeC,ZhangJ,ZhangH,WeiH,WuS,JiangW.
JImmunol.Jul15;(2):-.
17
CuttingEdge:IntestinalMucusLimitstheClonalDeletionofDevelopingTCellsSpecificforanOralAntigen.
最新消息:肠道粘液限制了口腔抗原特异性T细胞的克隆缺失。
TsaiK,HuangYH,MaC,BaldwinTA,HarderKW,VallanceBA,PriatelJJ.
JImmunol.Jul15;(2):-.
18
ExploitingPre-ExistingCD4+TCellHelpfromBacilleCalmette-GuérinVaccinationtoImproveAntiviralAntibodyResponses.
利用卡介苗疫苗中已有的CD4+T细胞来改善抗病毒抗体反应。
NgTW,WirchnianskiAS,WecAZ,FelsJM,JohndrowCT,SaundersKO,LiaoHX,ChanJ,JacobsWRJr,ChandranK,PorcelliSA.
JImmunol.Jul15;(2):-.
19
ImmuneAutoregulatoryCD8TCellsRequireIFN-γResponsivenesstoOptimallySuppressCentralNervousSystemAutoimmunity.
免疫自调节CD8细胞需要IFN-γ反应来抑制中枢神经系统自身免疫。
BoydenAW,BrateAA,StephensLM,KarandikarNJ.
JImmunol.Jul15;(2):-.
20
DepletionofNKCellsImprovesCognitiveFunctionintheAlzheimerDiseaseMouseModel.
在阿尔茨海默病小鼠模型中,NK细胞的耗竭改善了认知功能。
ZhangY,FungITH,SankarP,ChenX,RobisonLS,YeL,DSouzaSS,SalineroAE,KuentzelML,ChitturSV,ZhangW,ZuloagaKL,YangQ.
JImmunol.Jul15;(2):-.
21
HMGB1-NeutralizingIgMAntibodyIsaNormalComponentofBloodPlasma.
HMGB1中和IgM抗体是血浆中的正常成分。
GengY,MunirathinamG,PalaniS,RossJE,WangB,ChenA,ZhengG.
JImmunol.Jul15;(2):-.
22
Double-Hit-InducedLeukocyteExtravasationDrivenbyEndothelialAdherensJunctionDestabilization.
内皮细胞粘附连接不稳定导致白细胞外渗。
MorsingSKH,Al-MardiniC,vanStalborchAD,SchillemansM,BieringsR,VlaarAP,vanBuulJD.
JImmunol.Jul15;(2):-.
23
FCRL4IsanFcReceptorforSystemicIgA,butNotMucosalSecretoryIgA.
FCRL4是系统性IgA的Fc受体,而不是黏膜分泌型IgA。
LiuY,GoroshkoS,LeungLYT,DongS,KhanS,CampisiP,PropstEJ,WolterNE,GrunebaumE,EhrhardtGRA.
JImmunol.Jul15;(2):-.
24
IL-23PromotesaCoordinatedBCellGerminalCenterProgramforClass-SwitchRe
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